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Treatment of adult nonlymphoblastic leukemia (ANLL) with AMSA in combination with cytosine arabinoside (ARA C) and Thioguanine (TG) (AAT)
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This model recapitulates important features of human ALS including the degeneration of spinal motor neurons, muscle weakness, and paralysis, as well as histological features such as pathological inclusions in neurons and glia, some of which contain mutant Sod1 (Stieber et al., 2000